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small clinical studies report improved liver enzymes with IV/oral GSH in NAFLD/ALD, and derivatives such as S-allyl-glutathione improve fibrosis in animals [116, 117]
Lack of Nrf2 in this model also restored basal protein oxidation levels (measured by an increase in 4-hydroxynonenal modification, a byproduct of lipid peroxidation)
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Upon activation, they polarize into either M1 (pro-inflammatory) or M2 (anti-inflammatory) phenotypes, exerting contrasting effects on neuronal survival (365)
