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(PubMed) Squire L, Berg D, Bloom F, du Lac S, Ghosh A, Spitzer N
In conclusion, our results from this study indicate that enhanced function of Gpx4 retards motor neuron disease of SOD1 G93A mice, likely through a mechanism of suppressing ferroptosis of motor neurons
56 Studies have also validated the role of neutrophils in the pathophysiology of APAP hepatotoxicity and the release of IL-1, which is produced when caspase-1 cleaves pro-IL-1
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