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The ability of Fn to invade epithelial cells via FadA binding to CDH1 (E- cadherin) and to promote inflammation and oncogenic signalling has been demonstrated using HCT116 xenograft mice (73) and Fn infection in an Apc Min/+ mouse model lead to the generation of a proinflammatory microenvironment (59)

Osuka and Van Meir, 2017)

doi: 10.1007/s12291-021-00982-1

Therefore, the consistent beneficial effects of BPC 157 therapy exclude poorly controlled NO increase (and therefore the consequent chain of negative events) (see below, Section 3.2) and poorly controlled effects on the increase of NOS gene expression (NOS-1, NOS-2, and NOS-3 gene expression)