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During AD, an uncoupling between the insulin receptor (IR) and the insulin receptor substrate 1 (IRS1) occurs as a consequence of the downregulation of IR expression and/or the inactivation of IRS proteins leading to the inability of insulin to promote its normal cellular downstream outcomes

Recent research highlighted the significant role of long noncoding RNA (lncRNA) in the occurrence and progression of IBD [81, 82]

Recognition of these electrophysiological alterations is crucial, as they may precede malignant ventricular arrhythmias and SCD

Kelp density was higher than pre-harvesting and higher than unharvested sites [67]