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Herein, we have approached this by analyzing transcriptomic and metabolomic responses elicited in colon cancer cells that persist after exposure either to the CDK4/6i Palbociclib or the glutaminase inhibitor Telaglenastat (CB-839), or their combination, in vitro and in vivo
Glutatyon un belirgin etkileri olan durumlar;
International journal of molecular sciences, 22(12), 6179
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