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The CPT1A/c-Myc signaling axis reduces lipid peroxide generation in lung cancer cells by suppressing ACSL4 expression and concurrently enhances resistance to ferroptosis through stimulation of the NRF2/GPX4 antioxidant pathway [69]
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We speculated that the change in behavior was related to impairment of synaptic transmission resulting from alcohol-induced oxidative damage and mitochondrial energy depletion, leading to loss of neuronal activity
