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doi: 10.4037/ajcc2009792

The peptide works primarily by: Slowing gastric emptying to prolong satiety after meals Reducing food intake through central nervous system signaling Promoting satiety via actions at the area postrema in the brainstem Complementing insulin action without directly affecting insulin secretion For those exploring peptide research, understanding cagrilintide's unique mechanism is crucial for appreciating its potential applications in metabolic health studies

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IDH2 and IDH3 act in mitochondria, mainly in the TCA cycle
