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36 , 37 Finally, c-Myc expression is often elevated in the resistant fraction following cisplatin treatment, as it not only promotes survival through the acquiring of chemo-resistance genes and upregulation of DNA repair proteins, but also has been found to be stabilized in certain cancers by chemo-induced gain or loss of micro-RNAs (miRNAs, such as miR-145) that target c-Myc

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While increased NOX activity may drive ROS production in the setting of sufficient NADPH stores, other NADPH-independent ROS may represent a more abundant alternative source of oxidative stress in this model of G6PD-d