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Notably, different death pathways are not isolated from each other, but there is extensive cross-talk and pathway switching, which is particularly crucial for understanding COPD characterized by chronic inflammation and tissue remodeling ( Figure 1 Before discussing the specific roles of different PCD forms in COPD, we first outline their core molecular mechanisms, which are essential for understanding the specific activation of these death programs in the pulmonary pathological environment

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Even though the general results remain stable (i.e

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