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In particular, respiratory epithelial tissues have been identified to be more susceptible to the gas/vapor phase of smoke 46,47
This modified type II response, which promotes IgG4 production while suppressing IgE responses, potentially explains the low levels of IgE in EoE and why anti-IgE treatments are unsuccessful [130]
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Something similar might happen in the U.K
Electronic supplementary material Below is the link to the electronic supplementary material
