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Introduction The advent of incretin-based therapies, including glucagon-like peptide-1 receptor agonists (GLP-1RAs) and their combinations, as pharmacological treatments for obesity and type 2 diabetes, has sparked intense debate about their role relative to traditional lifestyle interventions such as exercise and dietary modification
GLP-1 medications slow digestion and reduce hunger
When oxidative throughput remains sustained while substrate availability declines, discrete rate-limiting biochemical nodes may emerge [76, 77]
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