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The DM control group consisted of patients with T2DM who were prescribed insulin or glucose-lowering drugs but not incretin-based drugs (GLP-1 RAs or DPP-4 inhibitors)
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Furthermore, a GLP-2 analogue with a single amino acid substitution [Gly2]-GLP-2 exhibited substantial resistance to DPP-4 cleavage and robust intestinotrophic activity in normal rats in vivo (60)
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