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Description
Glucagon signalling in hepatocytes suppresses intermediaries in fatty acid synthesis and prompts catabolism of FFAs, leading them to beta-oxidation and degradation, thereby diverting them from storage as triglycerides and eventual release in the form of VLDL [72]
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Isolation and characterization of a novel endogenous peptide ligand for the human APJ receptor
That glucose-dependent mechanism is the key distinction
