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Pre-treatment with a GLP1 receptor-blocking antibody neutralised the CNO-dependent improvement in glucose tolerance but did not affect the reduction in food intake, and an independent group of animals pair-fed to the CNO-treatment group demonstrated attenuated weight loss
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It reveals the underlying disease biology again: hunger rises satiety falls energy expenditure stays suppressed the brain becomes more vulnerable to food reward central adiposity starts returning This is why obesity must be discussed like other chronic diseases
Your metabolism depends on adequate hydration
