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We emphasize that SYGL-1 and LST-1 must also have FBF-independent functions, because the lst-1 sygl-1 phenotype is more severe than the fbf-1 fbf-2 phenotype [15, 18], and because single sygl-1 and lst-1 mutants enhance the fbf-1 fbf-2 phenotype [25

In rodents, GLP-1 has been shown to increase the biosynthesis of glucose transporter 2 (GLUT-2) and glucokinase [29]

But once metabolic pressure is relieved, it is often mobilised earlier than subcutaneous fat

U.S.: 71%), followed by metabolic health/diabetes