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Davies MJ, Bergenstal R, Bode B, Kushner RF, Lewin A, Skjoth TV, Andreasen AH, Jensen CB, DeFronzo RA
Briefly, FFAs can reduce the tyrosine phosphorylation of insulin receptor substrate-1 by activating the serine/threonine kinase, thereby aggravating IR (151)
Together, these changes underscore the therapeutic advantage of hydrogel-based delivery, with PNP formulations enhancing both the consistency and duration of GLP-1 receptor agonist action
