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Description
SUMMARYIncretin therapy is based on the antidiabetic actions of the incretin hormone GLP-1
SGLT2 inhibitors cause a carbohydrate deficit by glucosuria, which leads to an increased glucagon/insulin ratio, the metabolic shift from glucose to lipid utilization causing ketogenesis, and hence euglycemic DKA
Explore the current evidence on GLP-1 receptor agonists and rheumatoid arthritis, including anti-inflammatory effects, clinical research, and why these medications are not recommended for joint disease
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