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GLP-1 decreases food intake by acting on GLP1R in the hypothalamus and brainstem, and improves glycemic control by slowing gastric emptying, increasing glucose-stimulated insulin secretion, suppressing glucagon secretion and increasing -cell mass

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The escalating prevalence of CVD, coupled with increasing T2DM and obesity rates, makes it important to find new ways of reducing the residual cardiovascular risk in patients treated with conventional therapies, including statins, antihypertensives, and antiplatelet agents [8]

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