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Through HDAC6 inhibition, the pathway promotes the removal of damaged cellular components, reducing oxidative stress and enhancing cellular repair mechanisms in retinal cells ( 2.2.3 Beyond the eye In renal tubular cells ( + -dependent deacetylase SIRT1, reducing mitochondrial dysfunction and oxidative stress by decreasing ROS production, thereby preventing apoptosis induced by lipotoxicity

Blood was collected in K 2 EDTA tubes and kept on wet ice prior to 10 min centrifugation (3000 g ) at 5 C 3 C

These two Tyr residues in turn form pi-pi interactions