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This may explain why ATM-deficient cells and cells derived from patients with ataxia telangiectasia, who have a mutated inactive ATM gene, have high levels of ROS

Glutamate and cysteine form -glutamylcysteine, followed by the addition or conjugation of glycine to complete GSH synthesis (1-3)

Therefore, it is suggested that CD8 + T cell can initiate immunogenic cell death by inducing ferroptosis, and can amplify anti-cancer immune responses as well as broaden ideas for cancer therapy

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