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BPC 157-treated rats exhibited normal pupil diameter, microscopically well-preserved ganglion cells and optic nerve presentation, normal fundus presentation, normal retinal and choroidal blood vessel presentation, and normal optic nerve presentation [11]

These findings with catalase suggest that excess GPx-1 may potentiate apoptosis as a consequence of the disruption of normal (adaptive) oxidant signaling ( i.e ., a reductive stress leading to lack of survival) and not by other properties of GPx-1

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