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Patients with OA exhibit elevated iron levels in synovial tissue, and the impairment of iron homeostasis-regulating genes causes cellular iron accumulation, resulting in heightened production of ROS as well as the increased level of collagen II and MMP13, which are established hallmarks of OA [85] (Figure 2)
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Kim S, Jung UJ, Kim SR: Role of Oxidative Stress in BloodBrain Barrier Disruption and Neurodegenerative Diseases
