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Its dual mechanism as a GLP-1 receptor agonist provides powerful effects on both glycemic control and body weight regulation, with efficacy that exceeds previous pharmacological approaches
Mechanistically, stabilization of wild-type p53 and induction of the p53 target gene CDKN1A (p21) leads to decreased expression of the ribonucleotide reductase (RNR) subunits RRM1 and RRM2
