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(2024, October 15)
Targeting metabolic vulnerabilities in specific macrophage subsets, such as limiting ferroportin-mediated iron export in M2 TAMs or enhancing ACSL4-dependent lipid peroxidation in inflammatory TAMs, may shift the TME toward a ferroptosis-permissive state
The dosages for each peptide would remain within their typical ranges, but their combined effect is often greater than either alone
Biologics are made from living cells
