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Dysfunction in glycine receptor activity has been associated with epileptiform discharges, and it has been proposed that changes in GlyT1 expression within the hippocampus may underlie key aspects of the disorders neurochemical dysfunction, and the resulting imbalance in glycine homeostasis may contribute to the pathophysiology of epilepsy (Rigo et al

Particularly large amounts are generated in wounded and inflamed tissue through the respiratory burst of phagocytes
Protective effects of kahweol and cafestol against hydrogen peroxide-induced oxidative stress and DNA damage
