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Since researchers have proven alcohols inhibitory effect on glutamatergic neurotransmission through the NMDA receptors, many studies have tried to elucidate the molecular mechanisms underlying alcohols actions

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[41] [38] A recent study found that GATA1 is a strong enhancer of NBEAL2 expression and that the R216W and R216Q inactivating mutations in GATA1 may cause the development of alpha granule-deficient platelets by failing to stimulate the expression of NBDAL2 protein
