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To this end, there is a need to model demyelination and subsequent remyelination in animal models to enhance our understanding of oligodendrocyte susceptibility and to test potential pro-myelinating treatments that could be moved towards clinical therapies [22]
It also plays a role in glutathione synthesis, which supports detoxification pathways
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[DOI] [PMC free article] [PubMed] [Google Scholar] [59].Sweeney WE, Avner ED, Molecular and cellular pathophysiology of autosomal recessive polycystic kidney disease (ARPKD), Cell and Tissue Research, 326 (2006) 671685
