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Transcriptome-wide profiling of kidney biopsies and urinary sediment has defined gene-expression modules related to innate immunity, fibrosis and tubular dedifferentiation that stratify progression risk ( 5 Multi-omics signatures for risk stratification and patient subtyping Multi-omics approaches integrate genomic, transcriptomic, proteomic, metabolomic and, in some studies, epigenomic or spatial data from blood, urine and kidney tissue (Table 2

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