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miR-20a-5p mimics reduce renal I/R injury and ischemic postrenal fibrosis, while miR-20a-5p inhibitors have the opposite effect

Importantly, in vitro treatment with TNFi did not attenuate the cell death phenotype

Ox-LDL, oxidized low-density lipoprotein

Optimized immune function: NAD+ energizes immune cells while glutathione protects them from self-destruction during pathogen eliminationsupporting robust immune responses
