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Expression of this mutated gene greatly reduces expression and operation of astrocytic glutamate transporters, thus leading to an excitotoxicity linked to psychosocial abnormalities and impaired cognition

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In a rat model of trauma, GST content in the hippocampus showed time-dependent changes as early as 3 h post-trauma, accompanied by a synchronous increase in oxidative substances, and reached a peak at 24 48 h [33]