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BPC-157 complements this by mitigating drug-induced nephrotoxicity and ischemia-reperfusion damage, preserving kidney function through anti-apoptotic and anti-oxidative mechanisms

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In that regard, recent studies showed that FXR mediates an increased expression of Occludin, ZO-1, and claudin-1, all proteins involved in the formation of tight junctions, as well as defensins, involved in reducing the intestinal bacterial load [25]

10.5863/1551-6776-19.1.60 [DOI] [PMC free article] [PubMed] 17.Page MJ, McKenzie JE, Bossuyt PM et al (2020) statement: an updated guideline for reporting systematic reviews