iv glutathione and concussion Traumatic Brain Injury Antioxidant Therapies in Traumatic Brain
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10.1016/j.nut.2022.111655 173 OnakpoyaI
10.1016/j.ygeno.2021.06.013 98 KimB
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Addressing the mechanism of the H 2 O 2 -induced macrophage death, immunoblotting for cleaved caspase-3, measurement of released IL-1 and IL-18, and use of nercostatin-1 did not support caspase-3-dependent apoptosis, inflammasome activation or necroptosis as the underlying cause of the cytotoxicity, respectively
