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The uniform upregulation of important metabolites underscores a potential compensatory adaptation by the bacterium to maintain viability under the challenges of altered metabolic and oxidative states

Ethics declarations Ethics Approval Approval for this study was granted by the Ethics Committee of Mashhad University of Medical Sciences (941406)

Modification of substrate-inhibitor affinities of human platelet monoamine oxidase B in vitro

The LPS-TLR4-JNK/NF-B axis disrupts mitochondrial membrane potential and cristae architecture while downregulating Parkin expression, thereby blocking PINK1/Parkin-mediated mitochondrial autophagy