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Free radicals are responsible for inducing cellular damage and contributing to the process of aging

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Treatment Management of the patient with G6PD deficiency is determined by the clinical syndrome with which it is associated

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Results are not immediate but tend to become noticeable when the protocol is followed consistently under the guidance of a qualified provider who can monitor progress and adjust dosing as needed

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These changes are further intensified by acidic pH, divalent metal ions, and familial mutations (e.g., A53T and E46K), which disrupt droplet fluidity and enhance aggregation kinetics [177, 183]

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