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On top of that, during childhood and adolescence, individuals in the pre-schizophrenic phase show excessive loss of neuronal and synaptic connectivity to the extent that, during psychotic episodes, about one-third to one-half of patients show marked atrophic changes and enlargement of the lateral ventricles (33, 35)

These studies highlighted a potential mechanism by which SCs can actively modulate the local environment by influencing the biosynthesis of ECM components, further avoiding an excessive deposition of collagen during muscle adaptive responses (Figure 1b, lower panel)

RhoA/ROCK pathway plays a significant role in MetS-associated ED