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Treatment of A42-injected rats with an inhibitor of dipeptidylpeptidase-4, sitagliptin, together with quercetin, a flavonol from the flavonoid group of polyphenols, significantly diminished the level of A42 compared with either treatment alone but increased antioxidant protein levels (i.e., SOD, CAT) and the level of GSH and a diminution of histopathologically determined neuronal damage

Bafilomycin A1 disrupts autophagic flux by inhibiting both V-ATPase-dependent acidification and Ca-P60A/SERCA-dependent autophagosome-lysosome fusion

This lactate-NADH antioxidant mechanism operates in concert with the GSH system, collectively maintaining neuronal redox balance under conditions of activation or metabolic stress

Cocova-L 5000 IU is administered via intramuscular or subcutaneous injection