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Description
Beyond waste clearance, neurons and glia constantly exchange metabolic signals, with glial cells, particularly astrocytes, playing a critical role in redox homeostasis
Triggered by mitochondrial oxidative stress, Ca 2+ overload, dysregulation of iron homeostasis, mitochondrial DNA defects, and disruption of mitochondrial quality control (MQC), programmed cell death subsequently ensues encompassing apoptosis, pyroptosis, necroptosis, and ferroptosis [17]
When this balance is disrupted, hypertension occurs [96]
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