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Overexpression of PHB2 can activate mitophagy, restore mitochondrial activity, enhance myocardial cell viability, and inhibit endotoxin-induced mitochondrial dysfunction in myocardial cells [15]

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For more information about this incident and what Capital One is doing to respond, visit www.capitalone.com/facts2019

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This review article provides a comprehensive overview of the involvement of ROS in the pathophysiology of sepsis and the potential application of antioxidants with antimicrobial properties as an adjunct to primary therapies (fluid and antibiotic therapies) against sepsis

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