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A deeper understanding of the molecular pathways involving CTGF could facilitate the development of antifibrotic strategies, such as microRNA-26a and the use of CRISPR-Cas9 aimed at enhancing surgical success rates and minimizing complications (87, 89)
Retrieved from gov/articlerender.fcgi?artid=3698662&tool=pmcentrez&renderty pe=abstract 62
Homma H, Kurachi H, Nishio Y, et al
Durable remissions with ivosidenib in IDH1-mutated relapsed or refractory AML
