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Table 1 The most commonly used exogenous testosterone preparations and their derivatives in strength-based doping ( Legend, PO, per os (by mouth)

Experimental models demonstrate that high-dose testosterone administration increases hepatic reactive oxygen species production while suppressing endogenous antioxidant enzyme activity ( Intrahepatic cholestasis observed in anabolic steroid users has been linked to impaired bile-acid transport

Exp Clin Endocrinol Diabetes (2007) 115:5226

[19] has merit, as accumulation of acyl-CoAs can inhibit the activity of some tricarboxylic acid (TCA) cycle enzymes, adenine nucleotide translocation and ATP formation [8991]