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(2011) identified three mechanisms: BPC-157 increased fibroblast outgrowth from tendon explants in a dose-dependent manner, improved tendon cell survival under oxidative stress, and accelerated fibroblast migration through FAK-paxillin signaling

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[DOI] [PMC free article] [PubMed] [Google Scholar] 29.Pagnotta S, Tramutola A, Barone E, Di Domenico F, Pittal V, Salerno L, Folgiero V, Caforio M, Locatelli F, Petrini S, et al