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Since the ANKIB1-RBR but not its RING2 was required, we next generated ANKIB1-deficient cells expressing a RING1-mutant ANKIB1 (ANKIB1-RING1 mut ) with mutations in several conserved residues (C333A/I335A/C336A/C351A/H353A) and, indeed, TLR3 stimulation failed to activate TBK1 in these cells (Fig

10.1038/s41419-023-06342-6 151 SuiX.WangJ.ZhaoZ.LiuB.LiuM.LiuM.et al (2024)

Hence, it remains to be determined whether increases in circulating bilirubin predispose to or maybe increase sports performance

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