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In cerebral ischemiareperfusion injury, propofol treatment at a concentration of 200 mM inhibited mitochondrial permeability transition pore (mPTP) channels, and reduced calcium-induced mitochondrial swelling and the production of associated reactive oxygen species (ROS)
With the advent of new technologies and a deeper understanding of neuronal metabolism, the field is poised for significant breakthroughs that could transform our approach to treating neurological disorders (Lee and Silva, 2009)
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