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Autophagosomes can efficiently dispose of IAPP small oligomers in p62-containing vacuoles to keep them from becoming toxic (Gupta and Leahy, 2014)

Such a setup would precisely delineate the NSC-derived paracrine factors (e.g., BDNF, VEGF [18, 32], TGF-) responsible for suppressing microglial activation and promoting an anti-inflammatory phenotype, independently of in vivo complexities

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