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[DOI] [PubMed] [Google Scholar] 15.Shannon C.E., Ghasemi R., Greenhaff P.L., Stephens F.B

1m), indicating that KG-induced therapeutic resistance is not simply due to a CCNE1- or MYC-driven replication stress response

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This further leads to the reversal of mitochondrial dysfunction, thereby increasing mitochondrial -oxidation and reducing intracellular oxidative stress to prevent hepatic lipotoxicity (53, 54)
