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These findings are not contradictory to those in T cells (102) but rather reflect cell type-specific responses: SIN-induced T-cell apoptosis proceeds via caspase-3dependent mechanisms without Bcl-2 involvement, whereas in macrophages, apoptosis is mediated through ERK activation and modulation of Bcl-2 family proteins
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Cell-type-specific gene programs suggest immune and stromal drivers of relapse in colorectal cancer
