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10.1016/s0196-9781(98)00075-8 44 GeorgiadisD.DiveV
Exp Neurobiol 25:113 Rowland LP, Shneider NA (2001) Amyotrophic lateral sclerosis
Other works have shown that glutathione-ethyl ester is able to effectively prevent oxidative damage of mitochondrial complex I caused by ischemia/reperfusion and thereby reduces the infarct size and improved neurological outcome [34, 35]
The rate at which Fragment 176-191 caused AEs was not statistically different from the rate at which placebos caused AEs
