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[DOI] [PubMed] [Google Scholar] 103.Farvid M.S., Jalali M., Siassi F., Hosseini M
DJ-1 functions as a bona fide negative modulator of ferroptosis by sustaining S-adenosyl homocysteine hydrolase activity through altering its interaction with the negative regulator adenosylhomocysteinase like protein 1, thereby maintaining the transsulfuration pathway-mediated biosynthesis of homocysteine and subsequent GSH synthesis (157)
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(III) In the context of liver cancer, is it possible to activate ferroptosis selectively in hepatocellular carcinoma cells without affecting healthy cells
