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Brain 12 , 17 (2019)

Here, we showed that MG132 treatment effectively reversed Keap1-dependent Nrf2 degradation and the reduced expression of downstream antioxidant genes in HSV-1-infected cells and mice, indicating that the inhibition of Nrf2 degradation by MG132 contributes to the alleviation of HSV-1-induced ferroptosis and pathogenesis

Using the data from 19 case-controlled studies, 5 cross-sectional studies, and 3 prospective studies published before 1995, the meta-analysis of Boushey and colleagues (Boushey et al, 1995) relating basal hyperhomocysteinemia to atherosclerotic vascular disease indicates that, above 10 mol/L, a 5 mol/L increment in circulating homocysteine is associated with a significant increase in the risk of coronary artery disease (60% in men and 80% in women)

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