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Knockdown of NLRP3 or caspase-1 reduced neuronal death and reversed cognitive impairments [147], whereas PARP-1 -/- mice were observed to prevent synaptic damage, cognitive dysfunction, and microglial activation in an AD mouse model referring to the significant role of pyroptosis and parthanathos in the pathogenesis of AD [148, 149]

The compounds were characterized by NMR spectroscopy (Supplementary Fig

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Sema vs Tirz vs Reta (Comparison) The landscape of GLP-1based therapies continues to evolve rapidly, with each new compound building upon the mechanisms of its predecessors